Has known Side Effects
This is often the result of long or short term medication use. The same medication can have a range of side effects ranging from none at all to totally debilitating symptoms within different individuals. Reasons for this include individual genetics, individual detoxification capacity, nutrition status, duration of use and total number of medications being taken.
It becomes very difficult to establish clear causes of symptoms when multiple medications are being taken at once.
See SIDE EFFECTS LINKOUT at end of this profile.
No Nastiness Attributes Yet
This off course does not mean there aren't any. Toxno just hasn't found any yet. If you know of additional Nastiness Attributes or other Exposure Routes, please let us know. You can contact us , use the comment section on the profile pages or download our data contribution form. Please provide a source or a reference.
These attributes are ONLY based on peer-reviewed evidence. See link to Data Sources below. Everyone benefits from knowing this stuff. Please Share.
- CATEGORIES: Chemicals detected in flowback and produced water - collectively referred to as - hydraulic fracturing wastewater | Industrial/Workplace Toxin | Food Toxin | Natural Toxin | Animal Toxin | Indirect Additives Used in Food Contact Substances | A Hazardous Substance that may be found in the Australian Workplace | Medication Approved in USA
- SUBSTANCE LINEAGE: Inorganic Compounds | Homogeneous Metal Compounds | Homogeneous Alkali Metal Compounds | | Homogeneous Alkali Metal Compounds
- SYNONYMS: K | K+ | Kalium | Potassium (ion) | Potassium (K+) | Potassium alloy | Potassium cation | Potassium ion | Potassium ion (K+) | Potassium ion (K1+) | Potassium ion(+) | Potassium ion(1+) | Potassium monocation | Potassium(+) | Potassium(1+) | Potassium(1+) ion | Potassium(I) cation | UN1420 | UN2257
- DESCRIPTION: Has been used in CSG, Hydraulic Fracturing Operations (Fracking) as - Unknown | Potassium is an essential electrolyte. Potassium balance is crucial for regulating the excitability of nerves and muscles and so critical for regulating contractility of cardiac muscle. Although the most important changes seen in the presence of deranged potassium are cardiac, smooth muscle is also affected with increasing muscle weakness, a feature of both hyperkalaemia and hypokalaemia. Physiologically, it exists as an ion in the body. Potassium (K+) is a positively charged electrolyte, cation, which is present throughout the body in both intracellular and extracellular fluids. The majority of body potassium, >90%, are intracellular. It moves freely from intracellular fluid (ICF) to extracellular fluid (ECF) and vice versa when adenosine triphosphate increases the permeability of the cell membrane. It is mainly replaced inside or outside the cells by another cation, sodium (Na+). The movement of potassium into or out of the cells is linked to certain body hormones and also to certain physiological states. Standard laboratory tests measure ECF potassium. Potassium enters the body rapidly during food ingestion. Insulin is produced when a meal is eaten; this causes the temporary movement of potassium from ECF to ICF. Over the ensuing hours, the kidneys excrete the ingested potassium and homeostasis is returned. In the critically ill patient, suffering from hyperkalaemia, this mechanism can be manipulated beneficially by administering high concentration (50%) intravenous glucose. Insulin can be added to the glucose, but glucose alone will stimulate insulin production and cause movement of potassium from ECF to ICF. The stimulation of alpha receptors causes increased movement of potassium from ICF to ECF. A noradrenaline infusion can elevate serum potassium levels. An adrenaline infusion, or elevated adrenaline levels, can lower serum potassium levels. Metabolic acidosis causes a rise in extracellular potassium levels. In this situation, excess of hydrogen ions (H+) are exchanged for intracellular potassium ions, probably as a result of the cellular response to a falling blood pH. Metabolic alkalosis causes the opposite effect, with potassium moving into the cells. (A3431).
From Safe Work Australia and the Hazardous Substances Information System (HSIS) in Australia:
In contact with water releases flammable gases which may ignite spontaneously. Causes severe skin burns and eye damage | | A Hazardous Substance that may be found in the Australian Workplace. Check with your employer or health and safety officer. Stay informed and become aware of the dangers that surround you. This chemical is included on the list of recognised hazardous chemicals from the Safe Work Australia - Hazardous Substances Information System (HSIS) that is based on the Globally Harmonised System of Classification and Labelling of Chemicals (GHS)
Work Health and Safety (WHS) Regulations are the basis for hazardous chemicals regulations in Commonwealth, State and Territory jurisdictions in Australia. Under the model WHS Regulations, manufacturers and importers of substances, mixtures and articles supplied for use in workplaces are required to determine whether they are hazardous to health and safety before supply. The model WHS Regulations mandate that the hazards of a chemical as determined by the Globally Harmonised System of Classification and Labelling of Chemicals (GHS) must be included in safety data sheets and on labels. There are transitional arrangements in place for moving to the GHS-based system.
The GHS Hazardous Chemical Information List contains chemicals classified by an authoritative source (such as the European Commission or NICNAS) in accordance with the Globally Harmonized System of Classification and Labelling of Chemicals (the GHS). This list contains the vast majority of chemicals currently in HSIS. This list and its detail are regularly updated by Work Safe Australia. The model Work Health and Safety (WHS) Regulations require chemicals to be classified in accordance with the Globally Harmonised System of Classification and Labelling of Chemicals (GHS). However transitional arrangements allow use of classification information in HSIS derived from the Approved Criteria until the 31 December 2016.
- FORMULA: K
- DATA SOURCES: DATA SOURCES: T3DB | PubChem | EPA in USA | FDA Indirect Food Additives | Safe Work Australia - Hazardous Substances Information System (HSIS) | USA FDA APPROVED DRUG PRODUCTS
- LAST UPDATE: 28/04/2018
Mostly focused on Health Implications of Long Term Exposure to this substance
- POSSIBLE HEALTH CONSEQUENCES:
- ACTION OF TOXIN:
- TOXIN SITES OF ACTION IN CELL: "Cytoplasm", "Extracellular", "Golgi apparatus"
- Additional Exposure Routes: This is an endogenously produced metabolite found in the human body. It is used in metabolic reactions, catabolic reactions or waste generation. SEE MEDICATION SIDE EFFECTS
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